Abstract
Bilateral lesions of the hypothalamic ventromedial nucleus (VMN) effectively eliminated the induction by estrogen-progesterone of lordosis behaviors 4 to 6 weeks after the brain surgery. However, recovery of the lordosis response was clearly evident in the rats with effective VMN lesions 2 weeks after the first test. This suggests that the VMN may not be essential to the expression of hormonally induced lordosis behavior, but may be involved in a supplemental lordosis mediating mechanism.
Original language | English |
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Pages (from-to) | 595-600 |
Number of pages | 6 |
Journal | Experimental Neurology |
Volume | 68 |
Issue number | 3 |
DOIs | |
Publication status | Published - 1980 |
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ASJC Scopus subject areas
- Neuroscience(all)
- Neurology
Cite this
Recovery of sexual receptivity in female rats with lesions of the ventromedial hypothalamus. / Okada, Reiko; Watanabe, Hiroshi; Yamanouchi, Korehito; Arai, Yasumasa.
In: Experimental Neurology, Vol. 68, No. 3, 1980, p. 595-600.Research output: Contribution to journal › Article
}
TY - JOUR
T1 - Recovery of sexual receptivity in female rats with lesions of the ventromedial hypothalamus
AU - Okada, Reiko
AU - Watanabe, Hiroshi
AU - Yamanouchi, Korehito
AU - Arai, Yasumasa
PY - 1980
Y1 - 1980
N2 - Bilateral lesions of the hypothalamic ventromedial nucleus (VMN) effectively eliminated the induction by estrogen-progesterone of lordosis behaviors 4 to 6 weeks after the brain surgery. However, recovery of the lordosis response was clearly evident in the rats with effective VMN lesions 2 weeks after the first test. This suggests that the VMN may not be essential to the expression of hormonally induced lordosis behavior, but may be involved in a supplemental lordosis mediating mechanism.
AB - Bilateral lesions of the hypothalamic ventromedial nucleus (VMN) effectively eliminated the induction by estrogen-progesterone of lordosis behaviors 4 to 6 weeks after the brain surgery. However, recovery of the lordosis response was clearly evident in the rats with effective VMN lesions 2 weeks after the first test. This suggests that the VMN may not be essential to the expression of hormonally induced lordosis behavior, but may be involved in a supplemental lordosis mediating mechanism.
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UR - http://www.scopus.com/inward/citedby.url?scp=0018836504&partnerID=8YFLogxK
U2 - 10.1016/0014-4886(80)90113-2
DO - 10.1016/0014-4886(80)90113-2
M3 - Article
C2 - 7379896
AN - SCOPUS:0018836504
VL - 68
SP - 595
EP - 600
JO - Experimental Neurology
JF - Experimental Neurology
SN - 0014-4886
IS - 3
ER -