抄録
Skeletal muscle hosts all of the isoforms of nitric oxide synthase (NOS). It is well documented that nitric oxide (NO) regulates force generation and satellite cell activation, and therefore, damage repair of skeletal muscle. NO can also activate nociceptors of C-fibers, thereby causing the sensation of pain. Although delayed-onset of muscle soreness (DOMS) is associated with decreased maximal force generation, pain sensation and sarcomere damage, there is a paucity of research linking NO and DOMS. The present mini-review attempts to elucidate the possible relationship between NO and DOMS, based upon current literature.
本文言語 | English |
---|---|
ページ(範囲) | 89-94 |
ページ数 | 6 |
ジャーナル | Nitric Oxide - Biology and Chemistry |
巻 | 26 |
号 | 2 |
DOI | |
出版ステータス | Published - 2012 2月 15 |
外部発表 | はい |
ASJC Scopus subject areas
- 生化学
- 生理学
- 臨床生化学
- 癌研究