Nitric oxide: Is it the cause of muscle soreness?

Zsolt Radak*, Hisashi Naito, Albert W. Taylor, Sataro Goto

*この研究の対応する著者

研究成果: Review article査読

17 被引用数 (Scopus)

抄録

Skeletal muscle hosts all of the isoforms of nitric oxide synthase (NOS). It is well documented that nitric oxide (NO) regulates force generation and satellite cell activation, and therefore, damage repair of skeletal muscle. NO can also activate nociceptors of C-fibers, thereby causing the sensation of pain. Although delayed-onset of muscle soreness (DOMS) is associated with decreased maximal force generation, pain sensation and sarcomere damage, there is a paucity of research linking NO and DOMS. The present mini-review attempts to elucidate the possible relationship between NO and DOMS, based upon current literature.

本文言語English
ページ(範囲)89-94
ページ数6
ジャーナルNitric Oxide - Biology and Chemistry
26
2
DOI
出版ステータスPublished - 2012 2月 15
外部発表はい

ASJC Scopus subject areas

  • 生化学
  • 生理学
  • 臨床生化学
  • 癌研究

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